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Cell-cell Interactions During Pollination:
Fertilization in plants is remarkably species-specific; a series of cell-cell interactions enables female cells to selectively interact with appropriate pollen grains. During the past few years, our laboratory has identified many genes that control communication between pollen and the pistil cells. We have discovered mutations that define key stages in fertilization, from the adhesion of pollen grains to female cells (Zinkl et al., Development, 126: 5431-5440) to the targeting of pollen tubes to eggs — a process strikingly similar to axon guidance in animals (Wilhelmi and Preuss, 1996, Science 274: 1535-37). We are finding that these cell-cell interactions employ molecules that are quite different from those analyzed before, relying more often on lipohilic components than on protein-protein binding. We recently demonstrated that pollen adhesion depends on a species-specific lipophilic adhesive that generates a remarkably strong binding force between pollen grains and female cells (Zinkl et al., Development, 126: 5431-5440). This investigation has had an unexpected benefit — further analysis of the "molecular velcro" that mediates pollen adhesion promises to yield an array of molecules that bind in a dry environment in a selective manner. Other hydrophobic molecules embedded in
a lipophilic extracellular matrix mediate pollen recognition (Preuss et
al., 1993, Genes Dev. 7: 974-985), and lipids are necessary for the assembly
and function of hydrophobic proteins on the pollen surface (Mayfield and
Preuss, Nature Cell Biology 2: 128-130). Our work suggests that this
array of lipids and proteins together establish the initial interactions
that support pollen-pistil signaling; we are currently testing this hypothesis.
Even pollen tube guidance may depend on lipophilic cues — with the recent
cloning of a gene that mediates pollen tube targeting (Wilhelmi and Preuss,
in preparation) we have demonstrated an interesting connection to the biotinylated
enzymes that regulate synthesis of hydrophobic signaling molecules.
Future objectives:
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